Just like the sequel, there are strong traces of 2x multi-sampling AA on the 360, which masks the stair-stepping effect we see on high-contrast edges. This doesn't make an appearance on PS3, alas, though a post-processing pass on both platforms makes sure that jaggies are attended to, with the efficacy of this approach depending on how a scene is being lit. The net result is a game that looks smoother on 360, though the differences only become apparent in side-by-side shots.
On first glance, it appears the 360 offers a brighter and more vivid image, with heavier bloom effect on weapons and spotlights, plus a more saturated use of reds. The PS3 release appears much duller in direct comparison, but it's clear that the positioning of light sources - both indoors and out - has a more natural impact on the way shadows play across the terrain. This is easiest to pick out around globally illuminated areas, such as the very first visit to the Eden Prime colony which has rocky outcrops and city barriers casting shadows at angles relative to the sun's position. Curiously, the 360 version often omits shadowing around these elements at a distance.
Elsewhere, Mass Effect's motion blur effect has seemingly been removed outright on the PS3 version, while the 360 comes out in full force with both a camera and a subtle per-object implementation. This strikes us as very odd indeed, considering the option to toggle the blur on and off is available in the menus for each release. Regardless of what we do in the menus, it remains absent during both cut-scenes and gameplay in motion on Sony's platform. It may come as a relief to some that the intensity of 360's classic film grain filter has been greatly reduced on PS3 too.
By comparison, we're looking at a far smoother frame-rate on PS3 in likewise situations, usually holding at 30FPS where the 360 may fluctuate between numbers. It also happens to run without tearing a single frame, be it in cut-scenes or when running through a Citadel area packed with characters. Alas, there are still familiar weak points; drops to 15FPS are commonplace during sequences filled with transparent alpha effects, plus major hiccups down to 10FPS are possible when entering the Mako buggy. There also seems to be an issue with simply starting idle dialogue with squad members, which often causes a sharp stutter on each platform.
However, a gunfight in the Chora's Den night-club soon shows us there's little to celebrate on the performance side, where we experience a sustained stretch of play at 15FPS on both platforms. In practice, this choppy level of feedback makes aiming at enemies very tricky indeed, though the PS3 does offer some respite by not tearing through the frames it does render out. This goes from bad to worse during a shoot-out in the Mako buggy, which sees us stuttering down to 10FPS at the sight of snow trails, rocket explosions and artillery fire. It isn't a pretty sight on either console once alpha effects are involved, and it's disappointing to see such glaring issues cropping up in a game released at the tail end of the generation.
Battle with your comrades or even your own friends in this all-out galactic war to take Earth back. With co-op online multiplayer missions new to the Mass Effect universe , you can choose from a variety of classes and races, form an elite Special Forces squad, and combine weapons, powers and abilities to devastating effect as you all fight together to liberate key territories from enemy control in this third entry of the epic intergalactic RPG franchise.
Results: Of the 53 patients, six (11%) had isoattenuating tumors with a mean tumor-pancreas contrast of 9.25 HU +/- 11.3 during the pancreatic phase and 4.15 HU +/- 8.5 during the portal venous phase. The secondary signs of pancreatic tumor in these six patients included an interrupted pancreatic duct (n = 5), dilated biliary and pancreatic ducts (n = 1), atrophic distal pancreatic parenchyma (n = 3), and mass effect and/or convex contour abnormality (n = 3). The mean tumor-pancreas contrast for the remaining 47 patients was 74.76 HU +/- 35.61 during the pancreatic phase.
Conclusion: With no visible tumor-pancreas contrast for isoattenuating tumors, indirect signs such as mass effect, atrophic distal parenchyma, and an interrupted duct sign are important indicators for the presence of tumor.
CT can be normal as lesions are often isodense to normal brain parenchyma. There is a relative lack of mass effect and distortion compared to the extensiveness of involvement. There may be an ill-defined asymmetry or subtle hypoattenuation of the involved brain parenchyma.
Differentiation between tumor-like lesions and tumors of the CNS was essential for medical intervention strategy. First, intracranial tumors, the following types of tumors may display as mass lesions on MRI: high-grade gliomas, glioblastoma multiforme, and primary central nervous system lymphomas. The Ki-67 index for most tumors was high, but low for inflammation lesions. Furthermore, the atypical cells may be detected by CSF cytology in patients with intracranial tumors. The relative regional blood volume of high-grade gliomas in the periphery was increased, and most of lymphomas were isodense or hyperdense on CT, and slightly restricted on diffusion. Second, intracranial abscess, which has the following features: abscess revealed predominantly as round, closed-ring-like enhancement, and thicker part of the wall toward the direction of the cortex; restricted diffusion of the center part could be presented, with apparent diffusion coefficient diminished markedly; low relative regional blood volume of necrotic wall was a characteristic on perfusion imaging; in addition, increased lactate, amino acids, and a dual rim sign were displayed on MRS and susceptibility weighted imaging, respectively. Furthermore, constitutional features, such as fever, were the common concomitant symptom, and the lesion was reduced after several weeks of antibiotic treatment. Third, inflammatory pseudotumor, the most common neuroimaging manifestation, involved the brain infrequently. On MRI, iso- to hyperintense lesions were on T1, interdigitation with the adjacent cortex was on T2, and Cho peak was increased on MRS. Furthermore, PACNS should be distinguished from secondary vasculitis of the CNS related to rheumatological disorders such as Wegener granulomatosis, sarcoidosis, and Behçet disease. Our 2 patients did not have any skin lesions or orogenital ulcers, and the results were negative for serological tests of connective tissue disease and autoimmune diseases.
Go through the airlock into the Reaper itself. The Reaper activates its kinetic barriers. Joker radios that the Normandy cannot retrieve them until the barriers come down. EDI advises destroying the mass effect core to remove the barriers. However, the mass effect core also powers the mass effect fields that prevent the Reaper from falling into the brown dwarf.
As soon as you pass by an explosive crate, Husks and an Abomination appear. It is best to backpedal and to blow up the crates when Husks are nearby. Husks only have melee attacks, so it is wise to keep your distance. Also, Abominations do suicide charges and will explode upon death, no matter how they died. Throughout this level in certain areas, Husks spawn by climbing up over the sides of the walkways. There will also be numerous explosive items mainly explosive crates and containers. Use them wisely, because the husks can quickly overwhelm you and your squad. Using Overload on an explosive item produces a larger blast than just shooting it. Try to use Overload on an explosive item only when there is a mass of enemies nearby to maximize the effect.
Plain magnetic resonance imaging (MRI) showed an ill-defined T2 hyperintense and T1 isointense lesion measuring ~ 1.6x1.9x2 cm with perilesional oedema involving the pineal gland, extending into the tectal plate, the posterior aspect of midbrain, pons, medulla, right superior and middle cerebellar peduncles. There was no diffusion restriction. Few foci of blooming on gradient images were seen, which represented calcifications. The lesion was seen to cause significant mass effect on aqueduct of Sylvius resulting in gross dilatation of both lateral and third ventricles (Evans index: 0.53). There was no midline shift.
Another differential would be pineal parenchymal tumours of intermediate differentiation, which are a rare type of pineal parenchymal tumours. These have imaging patterns between pineoblastoma (a malignant pineal parenchymal tumour) and pineocytoma (a benign pineal parenchymal tumour).  Teratomas are multilocular heterogeneous masses containing lipid areas.
I hope all the game breaking bugs have been fixed finally in this release. And the difficulty in some spots has been adjusted. The last mass effect was a pure pain in this matter and the reason it was the most hated title in the franchise.
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Head CT scan may be helpful in the diagnosis of intracerebral metastases. On CT scan, intracerebral metastases are characterized by iso- to hypodense mass with zero to marked peritumoral edema. On contrast administration, variable enhancement (intense, punctuate, nodular, or ring-enhanced) may be present. 2b1af7f3a8